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Pinnacle Research Labs

SLUPP332

SLUPP332

$50.00 AUD
$99.95 AUD $50.00 AUD
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Studied for its role in: 

  • potential to enhance fatty acid oxidation and promote greater utilization of lipids as an energy source.
  • Studied for its ability to increase cellular energy expenditure through activation of metabolic signaling pathways.
  • Research suggests it may support mitochondrial function, contributing to more efficient cellular energy production.
  • Investigated for its potential to improve endurance-related metabolic adaptations by enhancing oxidative metabolism.
  • Studied as an exercise-mimetic compound, with research exploring its ability to activate pathways normally stimulated during physical activity.
  • Research is investigating its role in promoting metabolic flexibility, allowing cells to more efficiently switch between carbohydrate and fat utilization.

Product Description

SLU-PP-332: 1000mcg/ml (1mg/ml) - 30ml

How it works

SLU-PP-332 is currently being investigated as a potent synthetic agonist of estrogen-related receptors (ERRs), particularly ERRα, ERRβ, and ERRγ, orphan nuclear receptors extensively implicated in the transcriptional regulation of mitochondrial energetics, oxidative phosphorylation, and metabolic homeostasis. Activation of these receptors is believed to modulate the expression of genes involved in mitochondrial biogenesis, fatty acid β-oxidation, electron transport chain activity, and adaptive energy metabolism.

Preclinical investigations suggest that SLU-PP-332 may influence downstream signaling cascades associated with PGC-1α (Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha), a master regulator of mitochondrial proliferation and oxidative metabolic adaptation. Through potential upregulation of PGC-1α-mediated transcriptional networks, SLU-PP-332 is being explored for its capacity to enhance mitochondrial respiratory efficiency, increase oxidative muscle fiber phenotype expression, and promote elevated basal energy expenditure under experimental conditions.

Emerging research also indicates that ERR agonism may influence metabolic substrate partitioning by promoting preferential lipid utilization and increasing the transcription of genes associated with fatty acid transport, tricarboxylic acid (TCA) cycle flux, and ATP biosynthesis. Experimental models have additionally suggested potential interactions with AMPK-mediated energy sensing pathways and cellular endurance adaptation mechanisms.

COA (Certificate of analysis)

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Legal Disclaimer

Disclaimer: All products are sold for research purposes only. The available products are not intended for human consumption, personal use, or any form of medical application. These materials are strictly for laboratory research, scientific studies, and educational purposes only (e.g.., lab or university use). Pinnacle Research Labs is not a licensed pharmacy, medical facility, or compounding service, and does not offer products for medical or clinical applications. Pinnacle Research Labs has never and will never make health and/or therapeutic claims. Buy purchasing our products you are agreeing to our terms of services.

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